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Study 9 of 17HMG (Human Menopausal Gonadotropin) literaturePubMed · Meta-analysis2026

Recombinant follicle-stimulating hormone (rFSH) versus other recombinant or urinary gonadotropins for ovarian stimulation in assisted reproductive technology cycles.

Live birth rates may be lower and the risk of ovarian hyperstimulation syndrome may be higher with rFSH compared to HMG/HP-HMG in assisted reproductive technology cycles.

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Where it sits

this study against the rest of the hmg (human menopausal gonadotropin) corpus
0
Preclinical
13
Observational
0
Open-label
3
Randomised
1
Reviews · this one

Summary and findings

This review compares the effectiveness and safety of recombinant follicle-stimulating hormone (rFSH) with various gonadotropins in women undergoing IVF or ICSI. The analysis included 59 studies with a total of 18,119 participants. Key findings suggest that live birth rates may be lower with rFSH compared to HMG/HP-HMG.

How much of this paper we could read: full text read (0.90). We had a clear abstract, so the summary below closely tracks the paper. What this means →
Live birth is probably lower with rFSH compared to HMG/HP-HMG (OR 0.83, 95% CI 0.73 to 0.95; 15 studies, 4793 participants, moderate-certainty evidence).2026

Abstract

The authors’ words, as PubMed supplied them

<h4>Rationale</h4>Ovarian stimulation is an essential step during in vitro fertilisation (IVF) and intra-cytoplasmic sperm injection (ICSI), needed to produce multiple follicles for follicle aspiration. The strategy of stimulating the ovaries with gonadotropins is well established and can be performed using different types of gonadotropins. This review update brings together all randomised studies that compare recombinant follicle stimulating hormone (rFSH) with urinary gonadotropins and other recombinant rFSH (including rFSH from a human cell line) and biosimilars.<h4>Objectives</h4>To compare the effectiveness and safety of recombinant follicle-stimulating hormone (rFSH) with the three main types of urinary gonadotropins, rFSH derived from a human cell line (follitropin delta) and rFSH biosimilars for ovarian stimulation in women undergoing IVF or ICSI treatment cycles.<h4>Search methods</h4>We carried out an extensive search on 31 March 2025, according to Cochrane guidelines. The databases searched included: the Cochrane Gynaecology and Fertility Specialised Register of controlled trials, CENTRAL, MEDLINE and Embase. We also searched trials registries, reference lists and contacted experts in the field for additional studies.<h4>Eligibility criteria</h4>We included all randomised controlled trials reporting data comparing clinical outcomes of women undergoing IVF/ICSI cycles and using rFSH in comparison with human menopausal gonadotropin (HMG) or highly purified HMG (HP-HMG), purified urinary follicle-stimulating hormone (FSH-P), highly purified urinary FSH (FSH-HP), follitropin delta, and rFSH biosimilars for ovarian hyperstimulation. We used the Trustworthiness in RAndomised Controlled Trials (TRACT) checklist to assess trustworthiness and excluded potentially problematic studies from the analyses.<h4>Outcomes</h4>We implemented the core outcome set for infertility. Our main outcomes were live birth, ovarian hyperstimulation syndrome (OHSS) (both critical outcomes) and clinical pregnancy.<h4>Risk of bias</h4>We used the Cochrane risk of bias tool (RoB 1) to assess the risk of bias.<h4>Synthesis methods</h4>We combined data using a fixed-effect model to calculate an odds ratio (OR) or mean difference (MD). We summarised the overall certainty of evidence for the main outcomes using GRADE criteria.<h4>Included studies</h4>We included 59 studies (25 new studies) with a total of 18,119 women undergoing fertility treatment.<h4>Synthesis of results</h4>rFSH versus HMG/HP-HMG Live birth is probably lower with rFSH compared to HMG/HP-HMG (OR 0.83, 95% CI 0.73 to 0.95; 15 studies, 4793 participants, moderate-certainty evidence). OHSS is probably higher with rFSH compared to HMG/HP-HMG (OR 1.42, 95% CI 1.12 to 1.80; 37 studies, 9813 participants, moderate-certainty evidence). Clinical pregnancy is probably lower with rFSH compared to HMG/HP-HMG (OR 0.87, 95% CI 0.76 to 0.98; 15 studies, 4839 participants, moderate-certainty evidence). rFSH versus FSH-HP Live birth: there is probably little or no difference in live birth between rFSH and FSH-HP (OR 1.02, 95% CI 0.86 to 1.21; 15 studies, 2956 participants, moderate-certainty evidence) OHSS: there is probably little or no difference in OHSS between rFSH and FSH-HP (OR 1.01, 95% CI 0.67 to 1.53; 18 studies, 3640 participants, moderate-certainty evidence). Clinical pregnancy: there is probably little or no difference in clinical pregnancy between rFSH and FSH-HP (OR 1.00, 95% CI 0.88 to 1.14; 22 studies, 4535 participants, moderate-certainty evidence). rFSH versus follitropin delta Live birth: there is probably little or no difference in live birth between rFSH and follitropin delta (OR 0.88, 95% CI 0.74 to 1.04; 3 studies, 2688 participants, moderate-certainty evidence). OHSS is probably higher with rFSH compared to follitropin delta (OR 1.62, 95% CI 1.18 to 2.23; 4 studies, 2988 participants, moderate-certainty evidence). Clinical pregnancy: there is no difference in clinical pregnancy between rFSH and follitropin delta (OR 0.94, 95% CI 0.80 to 1.11; 3 studies, 2688 participants, high-certainty evidence). rFSH versus biosimilars Live birth is probably higher with rFSH compared to biosimilars (OR 1.29, 95% CI 1.09 to 1.54; 8 studies, 2883 participants, moderate-certainty evidence). OHSS: there may be little or no difference in OHSS between rFSH and biosimilars (OR 0.77, 95% CI 0.57 to 1.06; 8 studies, 2883 participants, low-certainty evidence). Clinical pregnancy was probably higher with rFSH compared to biosimilars (OR 1.35, 95% CI 1.14 to 1.60; 7 studies, 2783 participants, moderate-certainty evidence). The certainty of the evidence ranged from low to moderate for live birth and OHSS, and from moderate to high for clinical pregnancy. The main limitations were risk of bias in the included studies - unclear selection bias and selective reporting, as well as high risk of other bias. Many studies were sponsored by the pharmaceutical industry.<h4>Authors' conclusions</h4>Live birth and clinical pregnancies are probably lower with rFSH compared to HMG/HP-HMG, while OHSS is probably higher with rFSH when compared to HMG/HP-HMG. There is probably little to no difference in live birth, clinical pregnancy, or OHSS between rFSH and FSH-HP. When compared with follitropin delta, there is probably little or no difference in live birth or clinical pregnancy, while OHSS is probably higher with rFSH. When compared with biosimilars, live birth and clinical pregnancies are probably higher with rFSH, while there may be little or no difference in OHSS.<h4>Funding</h4>This Cochrane review had no dedicated funding.<h4>Registration</h4>Protocol (2005) https://doi.org/10.1002/14651858.CD005354 Review (2011) https://doi.org/10.1002/14651858.CD005354.pub2.

Background

The paper addresses the effectiveness of different gonadotropins, specifically rFSH, in ovarian stimulation for assisted reproductive technology (ART) cycles. Previous studies have shown varying results regarding the efficacy and safety of recombinant versus urinary gonadotropins. Understanding these differences is crucial for optimizing ART protocols and improving patient outcomes.

Methods

The study design is a systematic review of randomized controlled trials comparing rFSH with other gonadotropins. The population includes women undergoing ART, but the exact sample size (n) is not reported in the abstract. The duration and specific doses of gonadotropins administered are also not detailed.

Results

Not reported in abstract.

Interpretation

Without specific results, it is difficult to compare this study's findings with existing literature. If significant differences were found, it would be essential to evaluate their clinical relevance, particularly in terms of treatment outcomes. Potential confounding factors include the variability in study designs and patient populations across the included trials.

Key findings

  • Not reported in abstract.

Limitations

  • Not reported in abstract.

Elsewhere in the HMG (Human Menopausal Gonadotropin) corpus

BComparison between the efficacy of short and long GnRH-a protocols as a clinical outcome for viable pregnancies in women undergoing IVF/ICSI Cycles.biorxiv-preprint · 2021 · Clinical pregnancy rates: LAP 33.12% vs SAP 28.23%, p=0.001.HumanBRecombinant Follicular Stimulating Hormone Plus Recombinant Luteinizing Hormone Versus Human Menopausal Gonadotropins- Does the Source of LH Bioactivity Affect Ovarian Stimulation Outcome?biorxiv-preprint · 2021 · Mean number of mature oocytes retrieved: 10 ± 5.8 in rFSH+rLH vs 8.3 ± 4.6 in HP-hMG, p=0.01.HumanBUsing Letrozole Cotreatment With Progestin-Primed Ovarian Stimulation In Women With Polycystic Ovary Syndrome Treated For IVFbiorxiv-preprint · 2021 · 42.22% implantation rate in letrozole group vs. 34.69% in control group, p<0.05HumanBEvaluation of assisted reproductive technology treatment outcomes based on stimulation dosages and anti-Mullerian hormone levelsbiorxiv-preprint · 2022 · Not reported in abstract.HumanBHighly purified-hMG versus rFSH in ovarian hyperstimulation in women undergoing elective fertility preservationbiorxiv-preprint · 2023 · Peak estradiol level was significantly lower in the rFSH group (2547.18±1648.21 pg/mL) than in the hp-hMG group (3468.02±2497.69 pg/mL, P<0.001).HumanBA FSH-secreting pituitary adenoma discovered after ovarian hyperstimulation syndrome: a case report, illustrating pitfalls in the interpretation of serum FSH levelsbiorxiv-preprint · 2024 · Estradiol elevated at 737 pg/ml.Human