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Study 29 of 29Orforglipron (LY-3502970) literaturebiorxiv-preprint · RCT

<span data-teams="true">Efficacy and Safety of Oral Small-Molecule (Non-Peptide) GLP-1 Receptor Agonists in Type 2 Diabetes and Obesity: A Systematic Review of the Complete Orforglipron and Danuglipron Clinical Development Programs

Orforglipron shows a statistically significant reduction in HbA1c and body weight in type 2 diabetes, but the clinical relevance of these findings should be carefully considered.

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Where it sits

this study against the rest of the orforglipron (ly-3502970) corpus
8
Preclinical
6
Observational
1
Open-label
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Randomised · this one
7
Reviews

Summary and findings

This systematic review assessed the efficacy and safety of orforglipron and danuglipron in adults with type 2 diabetes and/or obesity. A total of 9,693 participants were included from 13 phase 2/3 randomized controlled trials. Orforglipron demonstrated a pooled HbA1c reduction of −1.14 percentage points versus placebo.

How much of this paper we could read: full text read (0.80). We had a clear abstract, so the summary below closely tracks the paper. What this means →
Pooled HbA1c reduction of −1.14 percentage points versus placebo (95% CI −1.25 to −1.02; I²=0%)

Abstract

The authors’ words, as biorxiv-preprint supplied them

<h4>Background: </h4> /Objectives: Orforglipron and danuglipron are the two most clinically advanced oral small-molecule (non-peptide) GLP-1 receptor agonists, sharing a common mechanism of action but markedly different regulatory outcomes — approval versus discontinuation. No existing review synthesizes their complete registrational evidence. <h4>Methods:</h4> We systematically searched PubMed, Embase, Cochrane CENTRAL, and Web of Science (inception to September 2026; PROSPERO-registered protocol) for phase 2/3 randomized controlled trials of orforglipron or danuglipron in adults with type 2 diabetes and/or obesity. Risk of bias was assessed with Cochrane RoB 2. Comparable trials were pooled using random-effects meta-analysis; clinically heterogeneous or non-comparable trials were synthesized narratively. <h4>Results:</h4> Of 505 records identified (286 after deduplication), 13 phase 2/3 randomized controlled trials met the registered eligibility criteria (10 orforglipron, 3 danuglipron); a further 17 reports (phase 1, mechanistic, or post hoc analyses of these trials) were retained only as contextual evidence. Of the 13 eligible trials, 9 (6 orforglipron phase 3, 3 danuglipron phase 2; N=9,693) were included in the quantitative synthesis, of which subsets sharing a common comparator were combined by random-effects meta-analysis (3 trials for HbA1c, 2 for body weight in type 2 diabetes); the remainder are reported as standalone estimates. Orforglipron produced a pooled HbA1c reduction of −1.14 percentage points versus placebo (95% CI −1.25 to −1.02; I²=0%) and superior glycemic control versus dapagliflozin and oral semaglutide. Weight loss varied by population: −9.10% in obesity without diabetes (single trial) and a pooled −6.54% in type 2 diabetes (95% CI −7.96 to −5.11; I²=49.8%, explained by background therapy). No hepatic safety signal was observed across 11,220 pooled orforglipron participants. Danuglipron showed comparable glycemic and weight efficacy but substantially higher, dose-dependent discontinuation (up to 72.7% in one arm), and its development was discontinued after a probable drug-induced liver injury case identified in a later, unpublished study. <h4>Conclusions:</h4> Despite a shared molecular target, orforglipron and danuglipron followed divergent clinical trajectories driven by differences in dosing regimen, tolerability, and hepatic safety rather than by pharmacodynamic efficacy, which was broadly comparable between the two molecules. These findings underscore that efficacy alone is an incomplete predictor of clinical success for oral small-molecule GLP-1 receptor agonists.

Background

Not reported in abstract.

Methods

Not reported in abstract.

Results

Not reported in abstract.

Interpretation

Not reported in abstract.

Limitations

Not reported in abstract.

Elsewhere in the Orforglipron (LY-3502970) corpus

AOrforglipron: An Oral GLP-1 Receptor Agonist for Obesity Treatment.The Annals of pharmacotherapy · 2026 · mean 7.1% to 10.6% placebo-adjusted weight reduction with orforglipronreviewBGLP-1 receptor agonists and ocular safety- Pharmacokinetic insights into ischemic optic neuropathy and diabetic retinopathy risk: A review.Biomolecules & biomedicine · 2026 · Hazard ratios for NAION ranged from 1.29 to 7.64.reviewAEfficacy and safety of orforglipron 12 mg versus orforglipron 36 mg maintenance dose among patients with obesity with or without diabetes: a systematic review and comprehensive meta-analysis.BMC endocrine disorders · 2026 · n=3459 · Percentage body weight change p < 0.00001.reviewAPredicting the Long-Term Risk of Type 2 Diabetes and Cardiovascular Disease With Orforglipron in People With Overweight or Obesity: A Post Hoc Analysis of the ATTAIN-1 Trial.Diabetes, obesity & metabolism · 2026 · -48.6% to -59.4% relative reduction in T2D risk score vs placebo (-10.5%; p<0.0001; HR-range 0.43-0.55)HumanDOrforglipron and the Kidney: A Promise or Disappointment? The Need for Trials Examining Renal Outcomes.American journal of nephrology · 2026 · Not reported in abstract.reviewBExploratory Cardiovascular Risk Modeling of Orforglipron Using the PREVENT Equations Compared with the Pooled Cohort Equations: A Post-Hoc Analysis from a Systematic Review and Meta-analysis.High blood pressure & cardiovascular prevention : the official journal of the Italian Society of Hypertension · 2023 · For a representative 50-year-old man with T2D at the 36 mg dose, PCE estimated a 10-year RRR of 16.8%, while PREVENT projected an RRR of 31.4%.review