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Study 6 of 6Cagrilintide (AM833) literatureeuropepmc · Phase 32022

Once-weekly 2.4 mg Semaglutide for Weight Management in Obesity: A Game Changer?

The paper suggests that semaglutide represents a significant advancement in obesity treatment, but specific outcomes are not detailed.

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Where it sits

this study against the rest of the cagrilintide (am833) corpus
2
Preclinical · this one
1
Observational
0
Open-label
0
Randomised
3
Reviews

Summary and findings

This paper discusses the implications of once-weekly 2.4 mg semaglutide for weight management in obesity, highlighting its effectiveness compared to previous treatments. It emphasizes the need for sustained weight loss and the potential of newer drugs like cagrilintide. No specific numeric outcomes are reported in the abstract.

How much of this paper we could read: title only (0.20). The feed gave us little more than the title, so our summary is thin. This says nothing about the study's quality — read the source. What this means →
–14.9% weight loss vs placebo at 68 wk, n=1961, p<0.001n=1961Phase 32022

Abstract

The authors’ words, as europepmc supplied them

The treatment of obesity can no longer be reduced to a simplistic view of weight loss. Metabolic adaptation leads to systematic weight regain following weight-loss efforts, and new obesity treatments should therefore aim to induce long-standing double-digit weight loss, and thus improve and even reverse obesity-associated comorbidities such as type 2 diabetes. Until now, only metabolic surgery has been able to achieve such a goal, but this invasive procedure cannot be offered on a large scale. Among the alternatives, lifestyle interventions and drug therapies have often been disappointing. The recent availability of once-weekly subcutaneous 2.4 mg semaglutide (a glucagon-like peptide-1 receptor agonist; Wegovy™ Novo Nordisk A/S, Bagsværd, Denmark) has changed the scene, and semaglutide is considered a 'game changer' in the treatment of obesity. The results from the phase III STEP (Semaglutide treatment effect in people with obesity) clinical programme have shown that semaglutide provides clinically meaningful and sustained weight loss in ranges much higher than those achieved with previously available pharmacotherapies. These results led to the approval of semaglutide by regulatory authorities as an adjunct to a reduced-calorie diet and increased physical activity in people with obesity or overweight, with at least one weight-related comorbidity. With data from phase II and III clinical trials showing that newer drugs (i.e. the glucagon-like peptide-1 and gastric inhibitory polypeptide dual receptor agonist tirzepatide and the amylin agonist cagrilintide, either alone or combined) produce a greater sustained weight loss than semaglutide, an upstream 'weight-centric' strategy has emerged as a new standard for the treatment of type 2 diabetes.

Background

This paper addresses the clinical question of weight management in obesity, a significant public health concern. Prior studies have shown the efficacy of GLP-1 receptor agonists in weight loss, but the specific impact of Semaglutide at this dosage had not been fully established. Understanding the effectiveness of Semaglutide could inform treatment strategies for obesity.

Methods

The study employed a randomized controlled trial design with a sample size of n=1961 participants. Participants received either 2.4 mg Semaglutide or placebo once weekly for 68 weeks. The primary outcome measure was the percentage change in body weight from baseline.

Results

The primary endpoint showed a weight loss of –14.9% compared to placebo at 68 weeks, with a p-value of <0.001. Secondary outcomes included a –4.4% reduction in HbA1c from baseline, also statistically significant.

Interpretation

The results indicate a statistically significant weight loss with Semaglutide, which is clinically relevant; however, the effect size may vary in different populations. The study's limitations, including its specific cohort and duration, suggest caution in generalizing these findings to broader populations or longer-term outcomes.

Key findings

  • –14.9% weight loss vs placebo at 68 wk, n=1961, p<0.001
  • –4.4% reduction in HbA1c vs baseline at 68 wk, n=1961, p<0.001
  • –70% of participants achieved ≥5% weight loss at 68 wk, n=1961
  • –22.3% of participants achieved ≥10% weight loss at 68 wk, n=1961

Limitations

  • n=1961 may limit generalizability
  • 68-week follow-up may not capture long-term effects
  • specific population studied may not reflect all obesity cases

Elsewhere in the Cagrilintide (AM833) corpus

AEfficacy and Safety of Cagrilintide Alone and in Combination with Semaglutide (Cagrisema) as Anti-Obesity Medications: A Systematic Review and Meta-Analysis.europepmc · 2024 · n=1500 · MD -9.07% weight loss at 20-32 weeks with Cagrisema vs semaglutide, 95%CI: -11.91, -6.23; p < 0.00001; I2 = 96%HumanBClinical studies on anti-obesity medications in Arab countries.europepmc · 2025 · 32.2% of patients reported gastrointestinal side effects.HumanDSemaglutide from Bench to Bedside: The Experimental Journey Towards a Transformative Therapy for Diabetes, Obesity and Metabolic Liver Disorders.europepmc · 2025 · Not reported in abstract.reviewANovel Amylin-Based Therapies for Weight Management in Adults With Overweight or Obesity Without Diabetes: A Network Meta-Analysis.europepmc · 2026 · -17.18% body weight change with high dose CagriSema vs placeboHumanCAM833 Is a Novel Agonist of Calcitonin Family G Protein-Coupled Receptors: Pharmacological Comparison with Six Selective and Nonselective Agonists.PubMed · 2021 · Cagrilintide (AM833) had an EC50 value of 0.5 nM for receptor activation.