Mechano growth factor E peptide promotes rat bone marrow-derived mesenchymal stem cell migration through CXCR4-ERK1/2.
MGF-25E promotes the migration of rat mesenchymal stem cells by activating the CXCR4-ERK1/2 signaling pathway, but the implications for human therapy remain unclear.
Where it sits
this study against the rest of the mgf (mechano growth factor) corpusSummary and findings
This study investigated the effects of Mechano Growth Factor E (MGF-E) on the migration of rat bone marrow-derived mesenchymal stem cells (rMSCs). The research focused on the modulation of the CXCR4-ERK1/2 signaling pathway and its impact on the mechanical properties of rMSCs. MGF-25E was found to increase stiffness and F-actin polymerization in rMSCs.
Abstract
Mechano growth factor (MGF) is a splicing variant of insulin-like growth factor 1 (IGF-1). The unique C-terminal E domain of MGF (MGF-E) makes it distinct from the other variants of IGF-1. Our previous work demonstrated that MGF-25E induces the migration of rat bone marrow-derived mesenchymal stem cells (rMSCs) by altering their mechanical properties, which is accompanied by the activation of the extracellular signal-regulated kinase 1/2 (ERK1/2) pathway. However, the relationship between ERK1/2 activation and the change in mechanical properties has not been illustrated. In the present study, we determined that MGF-25E induced the migration of rMSCs by modulating CXCR4 to activate the ERK1/2 pathway. The analysis of the Young's modulus and F-actin remodeling indicated that MGF-25E increased the stiffness and the F-actin polymerization of rMSCs through the activation of the CXCR4-ERK1/2 pathway. For the first time, this study clarified the signaling pathway that regulates the mechanical properties of rMSCs and is responsible for MGF-25E-promoted migration.
Background
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Methods
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Results
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Interpretation
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Key findings
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Limitations
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