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Study 13 of 15Pasireotide literaturebiorxiv-preprint · Observational2025

Multi-Layered Molecular Profiling Informs the Diagnosis and Targeted Therapy of Desmoplastic Small Round Cell Tumor

Multi-omics profiling can inform treatment decisions in desmoplastic small round cell tumor, with a 62% disease control rate observed in patients receiving recommended therapies.

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Preclinical
10
Observational · this one
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Summary and findings

This study analyzed molecular profiles in 30 refractory desmoplastic small round cell tumor (DSRCT) patients to inform treatment options. Molecular-based management recommendations were made for 28 patients (93%), with 13 receiving recommended therapies. Disease control was achieved in 8 patients (62%) with partial or stable responses.

How much of this paper we could read: full text read (0.80). We had a clear abstract, so the summary below closely tracks the paper. What this means →
62% disease control rate among patients receiving recommended therapies.2025

Abstract

The authors’ words, as biorxiv-preprint supplied them

<title>Abstract</title> <p> Desmoplastic small round cell tumor (DSRCT) is an ultra-rare sarcoma with limited treatment options. We performed whole-genome/exome, transcriptome, and DNA methylome analysis in 30 refractory DSRCT patients, complemented by (phospho)proteomic profiling in nine, within a nationwide precision oncology program. In eight patients (27%), DSRCT was diagnosed based on molecular profiling. Although all patients had “quiet” genomes, 28 (93%) received 107 molecular-based management recommendations, including assessment of clinical trial eligibility in 17 (57%). Nearly half of recommendations (45%) were based on overexpression of tyrosine kinases, as well as SSTR3/5 and CLDN6, detected in 33% and 20% of cases, respectively. Thirteen patients (46%) received recommended therapies, yielding disease control in eight (62%; partial response, <italic>n</italic>  = 5; stable disease, <italic>n</italic>  = 3), including three long-lasting responses (≥ 12 months) to pazopanib and trastuzumab deruxtecan, triggered by ERBB2 overexpression in the absence of constitutive ERBB2 signaling. Thus, multi-omics profiling enables individualized DSRCT treatment. </p>

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