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Study 2 of 6Octreotide literaturebiorxiv-preprint · Observational2026

Elimination Capacity Determines Outcome in Amatoxin Mushroom Poisoning-Induced Acute Liver Failure: A Globally Applicable Management Framework

The study reports a 98.8% transplant-free recovery rate among patients adhering to a new management protocol for amatoxin mushroom poisoning, highlighting the potential role of hydration and silibinin.

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Preclinical
5
Observational · this one
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Open-label
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Summary and findings

This study measured the outcomes of a management framework for amatoxin mushroom poisoning in a multicenter trial involving 99 patients. The intervention included sustained hydration, fasting plus octreotide, and intravenous silibinin. The study reported a transplant-free recovery rate of 98.8% among protocol-adherent patients.

How much of this paper we could read: full text read (0.80). We had a clear abstract, so the summary below closely tracks the paper. What this means →
Transplant-free recovery: 98.8% among protocol-adherent patients (n=86).2026

Abstract

The authors’ words, as biorxiv-preprint supplied them

<h4>ABSTRACT</h4> Amatoxin mushroom poisoning causes acute liver failure; progression from cholera-like hypovolemia resolution to fatal multisystem collapse occurs unpredictably. We conducted a prospective single-arm multicenter trial (N=99) investigating proactive toxicokinetic-based management: sustained hydration for renal protection and clearance preservation, fasting plus octreotide to arrest enterohepatic circulation, and intravenous silibinin to block hepatic reuptake. Transplant-free recovery: 98.8% among protocol-adherent patients (n=86). Multivariable analysis: sustained hydration (P<0.001) and silibinin (P=0.003) predicted survival; octreotide reduced coagulopathy recovery time (P=0.01); N-acetylcysteine and charcoal achieved neither. Conventional MELD score–outcome relationships vanished in protocol-adherent patients, suggesting management modified the natural history. Poor outcomes followed a reproducible sequence—hydration withdrawal, lactate rebound, oliguric nephropathy, multisystem failure—kidney failure, not liver, precipitated collapse. These findings establish renal clearance preservation and gallbladder sequestration as central outcome determinants, supporting a globally applicable management framework substituting gallbladder drainage when silibinin is unavailable or likely to fail. ClinicalTrials.gov identifier: NCT00915681 .

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