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Study 23 of 23NAD+ (Nicotinamide Adenine Dinucleotide) literaturebiorxiv-preprint · Observational2026

Metabolite-specific Reproducibility of Cerebral <sup>31</sup> P-MRS at 3T: Recommendations for Clinical Research

This study highlights the variability in 31P-MRS measurements of brain metabolites, emphasizing the need for careful planning in clinical trials to ensure reliable results.

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Where it sits

this study against the rest of the nad+ (nicotinamide adenine dinucleotide) corpus
7
Preclinical
14
Observational · this one
0
Open-label
1
Randomised
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Summary and findings

This study assessed the reproducibility of phosphorus magnetic resonance spectroscopy (31P-MRS) in measuring brain metabolites among healthy individuals and those with Parkinson's disease. It reported test-retest variability of metabolite ratios and provided sample-size recommendations for future studies. Notably, longer scan intervals increased intersession variability.

How much of this paper we could read: full text read (0.70). We had a clear abstract, so the summary below closely tracks the paper. What this means →
Test-retest variability ranged from ∼5-25 CoV%, n=Not reported in abstract.2026

Abstract

The authors’ words, as biorxiv-preprint supplied them

Phosphorus magnetic resonance spectroscopy ( 31 P-MRS) enables non-invasive measurement of brain metabolism, yet its reproducibility in clinical settings remains unclear. We systematically assessed intra- and intersession variability as well as inter-individual differences of key phosphorus metabolites at 3 Tesla in healthy individuals and persons with Parkinson’s disease under various experimental condition. Intersession variability, as measured by coefficients of variation (CoV) increased notably for longer scan intervals (∼1 year), and metabolite ratios from well-resolved spectral signals (i.e., adenosine triphosphate (ATP), phosphocreatine (PCr), intracellular inorganic phosphate P i ) exhibited consistently higher stability compared to ratios calculated from metabolite signals overlapping on the spectrum (e.g., total nicotinamide adenine dinucleotide (tNAD), as well as phosphate monoesters (PMEs) and phosphate diesters (PDEs). Test-retest variability ranged from ∼5-25 CoV%, where PCr, ATP-α and ATP-γ were the most stable while glycerophosphocholine (GPC), glycerophosphoethanolamine (GPE), phosphoethanolamine (PE) and tNAD varied considerably. Inter-individual variability was found to be higher than intra-individual variability for all metabolite ratios, ranging from ∼9-33 CoV%. By systematically quantifying intra-individual and inter-individual variability, as well as providing explicit sample-size recommendations, this study facilitates more reliable longitudinal and cross-sectional clinical trials and translational studies of brain metabolism featuring 31 P-MRS.

Elsewhere in the NAD+ (Nicotinamide Adenine Dinucleotide) corpus

CAging disrupts cumulus-oocyte NAD homeostasisbiorxiv-preprint · 2023 · n=30 · Not reported in abstract.mixedCThe placental metabolic clock.Science (New York, N.Y.) · 2026AnimalDMitochondrial quality control in human ageing and longevity.Nature metabolism · 2026DBacteria sense virus-induced genome degradation via methylated mononucleotides.Science (New York, N.Y.) · 2026 · Not reported in abstract.CMechanisms of apoptosis in colon cancer cells induced by IDO1 inhibitor and irradiation: A metabolomic studybiorxiv-preprint · 2026 · NAD+ levels decreased by additional 1-MT in radiated Colon26 cells (452 vs. 1027 nmol/g, p = 0.0495)AnimalCNicotinamide Riboside Enhances Mitochondrial Bioenergetics and Dopaminergic Signaling Independent of Neuron Survival in a Double-Hit Parkinson’s Modelbiorxiv-preprint · 2026 · n=20 · Not reported in abstract.Animal